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and phasic and tonic myotatic re exes But isolated neurons also become hypersensitive to neurotransmitters Since the early experiments of Cannon and Rosenblueth it has been known that section of sympathetic motor bers leaves the denervated structures hypersensitive to epinephrine; these authors also found the motor neurons in the isolated spinal segments to be abnormally sensitive to acetylcholine Various combinations of residual de cits (of lower and upper motor neurons and sensory neurons) are to be expected Some of the resulting clinical pictures are complete or incomplete voluntary motor paralysis; a accid atrophic paralysis of upper limb muscles (if appropriate segments of gray matter are destroyed) with spastic weakness of the legs; a partial or rarely a complete Brown-Sequard syndrome (pages 51 and 140); and each of these occurs with variable sensory impairment in the legs and arms High cervical lesions may result in extreme and prolonged tonic spasms of the legs due to release of tonic myotatic re exes Under these circumstances, attempted voluntary movement may excite intense contraction of all exor and extensor muscles lasting for several minutes Segmental damage in the low cervical or lumbar gray matter, destroying inhibitory Renshaw neurons, may release activity of remaining anterior horn cells, leading to spinal segmental spasticity Any residual symptoms persisting after 6 months are likely to be permanent, although in a small proportion of patients some return of function (particularly sensation) is possible after this time Loss of motor and sensory function above the lesion, coming on years after the trauma, occurs occasionally and is due to an enlarging cavity in the proximal segment of the cord (see further on, under Syringomyelia ) Transient Cord Injury (Spinal Cord Concussion) These terms refer to a transient loss of motor and/or sensory function of the spinal cord that recovers within minutes or hours but sometimes persists for a day or several days In most instances the symptoms are already diminishing and few neurologic abnormalities are found at the time of the rst examination There are a number of such transient syndromes; bibrachial weakness; quadriparesis (occasionally hemiparesis); paresthesias and dysesthesias in a similar distribution to the weakness; or sensory symptoms alone ( burning hands syndrome ) In the rst and last of these the central gray matter of the cervical cord is implicated It is assumed that the cord undergoes some form of elastic deformation when the head is struck at the vertex or frontally and the cervical spine is compressed or hyperextended; however, the same effects can be produced by direct blows to the spine or forceful falls at on the back and occasionally by a sharp fall on the tip of the coccyx Little is known of the pathologic changes or the mechanisms that underlie these reversible syndromes Spinal cord concussion from direct impact is observed most frequently in athletes engaged in contact sports (football, rugby, and hockey) An incomplete and reversible myelopathy occurs referable to the level of the injury A congenitally narrow cervical canal is thought to predispose to spinal cord concussion and to increase the risk of recurrence As with cerebral concussion, particularly if there have been previous concussions, a dif cult decision arises whether to allow resumption of competitive sports There are no reliable data on which to base this decision only guidelines that favor continued participation if the de cit has been brief Certainly, it is advisable in most cases to be certain that spinal instability has not been induced by the injury This can be ascertained by obtaining exion and extension radiographs of the af-. how to create barcodes in excel 2013 free Excel 97, 2000, XP, 2003 - Códigos de barras en Excel
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